Sunday, June 15, 2008

CrossFit Sydney Research

CrossFit Conditioning
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Cook&Phillip Aquatic Centre CBD
Sport or Goal Specific PT Sessions
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Contact
Andrew@CrossFitSydney.com.au






CFHQ WOD

"MICHAEL"

FOR TIME
3RDS
800M
50 GHD SIT UPS
50 BACK EXTENSIONS

RECORD TIME TAKEN


ENDURANCE WOD

Swim: 1000m/y at 80% for first 500m/y then pick it up to 90% or more on the last 500m/y

Bike: 20M at 80% for first 10M then pick it up to 90% or more on the last 10M

Run: 10k at 80% for first 5k then pick it up to 90% or more on the last 5k

C2: 2 x 1500m, go 80% first 1500m recover 2min, then 90% or more for second 1500m

STRENGTH WOD

SNATCH
3 ATTEMPTS AT 1RM

C&J
3 ATTEMPTS AT 1RM

BACK SQUAT
3 ATTEMPTS AT 1 RM



The Impact of Metabolic Stress on Hormonal Responses and Muscular Adaptations



Kazushige Goto; Naokata Ishii; Tomohiro Kizuka; Kaoru Takamatsu
Author Information

Purpose: The purpose of this study was to examine the impact of exercise-induced metabolic stress on hormonal responses and chronic muscular adaptations.
Methods: We compared the acute and long-term effects of an NR regimen (no-rest regimen) and those of a WR regimen (regimen with rest period within a set). Twenty-six male subjects were assigned to either the NR ( N = 9), WR ( N = 9), or control (CON, N = 8) groups. The NR regimen consisted of 3-5 sets of 10 repetitions at 10-repetition maximum (RM) with an interset rest period of 1 min (lat pulldown, shoulder press, and bilateral knee extension). In the WR regimen, subjects completed the same protocol as the NR regimen, but took a 30-s rest period at the midpoint of each set of exercises in order to reduce exercise-induced metabolic stress. Acute hormonal responses to both regimens were measured followed by a 12-wk period of resistance training.

Results: Measurements of blood lactate and serum hormone concentrations after the NR and WR regimens showed that the NR regimen induced strong lactate, growth hormone (GH), epinephrine (E), and norepinephrine (NE) responses, whereas the WR regimen did not. Both regimens failed to cause significant changes in testosterone. After 12 wk of resistance training, the NR regimen caused greater increases in 1RM ( P < 0.01), maximal isometric strength ( P < 0.05), and muscular endurance ( P < 0.05) with knee extension than the WR regimen. The NR group showed a marked increase ( P < 0.01) in muscle cross-sectional area, whereas the WR and CON groups did not.

Conclusion: These results suggest that exercise-induced metabolic stress is associated with acute GH, E, and NE responses and chronic muscular adaptations following resistance training.

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